shenyang myasthenia gravis
institution founded in may 2001, composes of clinics and in-patient
department, which combined research and treatment together.
it focuses on pathogeny, pathology, herbalist treatment principle,
and treatment methods of the nervous system disease. further
sum up over ten years experiences of president wan xiangcun
on treatment of myasthenia gravis and nervous system disease,
and based on “treatment of the combination of injection and
medicine”, and blazed a new path for completely curing this
world big problem--the nervous system disease. at present,
all patients had got completely well after treatment in our
institution.
myasthenia gravis,mg
pathogeny of mg
external cause—environment factor
on the base of inheritance infectious diathesis, incidence
by certain types of genetic defect could be induced by some
environment factors, like, virus infection, or usage of aminoglycoside
antibiotics or penicillamine medicines and etc.
internal cause—inheritance
recently there are many discoveries on autoimmune disease
research that it not only relates with gene of major histocompatibility
antigen complex, but relates with non-genes, like, t cell
receptor (tcr), immunoglobulin (ig), cell factor, apoptosis
gene, and other genes
based on the above statement:
this kind of autoimmune disease—myasthenia gravis is accrued
on the base of inheritance, which could be served as internal
cause; on external cause, it has been generally considered
that there is a relationship with chronic virus infection
on thymus
symptoms of mg
clinical characteristics: usually there are pathological changes
on extraocular muscle, facial muscle, muscle groups (of pharynx,
throat, palatine, tongue) which dominated by medulla oblongata,
neck muscle and scapular girdle muscle, respiratory muscle.
and the basic phenomena of the muscle pathological changed
due to pathological tiredness, namely, appearing temporary
weakness and even paralysis after many times action, turning
better after short rest, symptom fluctuation each days, light
in morning and serious at night. there are serious movement
disorders among terminal patients, and cannot recover completely
even after rest. adult patients of mg often have expressions
of ptosis and double vision. with the disease progressing,
they appear dyslalia, choke when feeding, bend neck, disability
for rise their heads, limbs weak and so on. some other patients
are with muscle atrophy on extraocular muscle, tongue muscle,
scapular muscle, triceps blachii muscle, quadriceps femoris
and etc.. children of mg mostly within the limit of extroacular
muscle, seldom tired their general skeletal muscles.
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